Akt-话题@MedSci

#Akt#

70篇内容 | 2940人围观

关注话题

话题活跃用户

#插入话题

插入图片

最新最热

[GetTopicListResponse(id=1656170, encodeId=779d16561e0ee, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=J Periodontol：TLR激活通过Akt信号通过Myd88或TRIF依赖性方式抑制人牙周膜干细胞的成骨潜能, objectType=article, longId=152581, objectId=6610152581fa, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=6610152581fa, replyNumber=0, likeNumber=55, createdTime=2019-07-30, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=6610152581fa, moduleTitle=J Periodontol：TLR激活通过Akt信号通过Myd88或TRIF依赖性方式抑制人牙周膜干细胞的成骨潜能, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=6610152581fa)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656172, encodeId=6d0316561e26a, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Clin Exp Pharmacol Physiol：MicroRNA let-7c-5p通过HMGA2/PI3K/Akt信号阻断抑制脂多糖诱导的炎症, objectType=article, longId=156806, objectId=6259156806cb, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=6259156806cb, replyNumber=0, likeNumber=67, createdTime=2019-01-09, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=6259156806cb, moduleTitle=Clin Exp Pharmacol Physiol：MicroRNA let-7c-5p通过HMGA2/PI3K/Akt信号阻断抑制脂多糖诱导的炎症, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=6259156806cb)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656175, encodeId=b18116561e5cd, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=J Cell Physiol：细胞外酶促合成的糖原通过Akt / GSK-3β信号传导途径激活成骨细胞分化来促进成骨, objectType=article, longId=157649, objectId=f13915e64942, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=f13915e64942, replyNumber=0, likeNumber=58, createdTime=2019-09-02, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=f13915e64942, moduleTitle=J Cell Physiol：细胞外酶促合成的糖原通过Akt / GSK-3β信号传导途径激活成骨细胞分化来促进成骨, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=f13915e64942)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656168, encodeId=a6571656168f0, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Cell Physiol Biochem：IL-8通过CXCR2介导的PI3k/Akt信号通路增强BMSCs的骨再生作用, objectType=article, longId=144753, objectId=3bf1144e5387, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=3bf1144e5387, replyNumber=0, likeNumber=62, createdTime=2018-12-28, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=3bf1144e5387, moduleTitle=Cell Physiol Biochem：IL-8通过CXCR2介导的PI3k/Akt信号通路增强BMSCs的骨再生作用, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=3bf1144e5387)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656163, encodeId=b0c71656163ef, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=J Clin Invest：PTEN/AKT和Hippo/YAP/TAZ通路之间的交叉串扰加速了NAFLD和肝癌的发生, objectType=article, longId=129225, objectId=2169129225fd, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=2169129225fd, replyNumber=0, likeNumber=54, createdTime=2018-04-01, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=2169129225fd, moduleTitle=J Clin Invest：PTEN/AKT和Hippo/YAP/TAZ通路之间的交叉串扰加速了NAFLD和肝癌的发生, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=2169129225fd)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656167, encodeId=cfaf165616e0b, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Blood：IGF-1通过激活Akt促进血小板生成, objectType=article, longId=139272, objectId=d1291392e224, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=d1291392e224, replyNumber=0, likeNumber=70, createdTime=2018-07-27, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=d1291392e224, moduleTitle=Blood：IGF-1通过激活Akt促进血小板生成, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=d1291392e224)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656162, encodeId=7bbf165616270, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Prostate Cancer and P D：生殖细胞中 IL4、MGMT和AKT1变异与前列腺癌特异性死亡率相关, objectType=article, longId=126609, objectId=9f1812660986, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=9f1812660986, replyNumber=0, likeNumber=86, createdTime=2018-11-10, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=9f1812660986, moduleTitle=Prostate Cancer and P D：生殖细胞中 IL4、MGMT和AKT1变异与前列腺癌特异性死亡率相关, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=9f1812660986)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656159, encodeId=ed3616561597e, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Int J Oncol：金-大黄酚纳米颗粒可通过失活AKT并诱导细胞凋亡和ROS产生抑制前列腺癌的进展, objectType=article, longId=122207, objectId=fe4212220e41, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=fe4212220e41, replyNumber=0, likeNumber=61, createdTime=2018-07-26, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=fe4212220e41, moduleTitle=Int J Oncol：金-大黄酚纳米颗粒可通过失活AKT并诱导细胞凋亡和ROS产生抑制前列腺癌的进展, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=fe4212220e41)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656160, encodeId=7afc165616085, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=CLIN CANCER RES：TMCO1影响膀胱尿路上皮癌AKT信号通路, objectType=article, longId=124543, objectId=b9ac1245430b, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=b9ac1245430b, replyNumber=0, likeNumber=65, createdTime=2018-12-03, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=b9ac1245430b, moduleTitle=CLIN CANCER RES：TMCO1影响膀胱尿路上皮癌AKT信号通路, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=b9ac1245430b)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656158, encodeId=4f17165615870, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=CLIN CANCER RES：AKT抑制剂MK-2206联合阿那曲唑治疗Ⅱ/Ⅲ期PIK3CA突变ER阳性和HER-2阴性乳腺癌, objectType=article, longId=120634, objectId=1dba1206345c, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=1dba1206345c, replyNumber=0, likeNumber=75, createdTime=2017-12-15, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=1dba1206345c, moduleTitle=CLIN CANCER RES：AKT抑制剂MK-2206联合阿那曲唑治疗Ⅱ/Ⅲ期PIK3CA突变ER阳性和HER-2阴性乳腺癌, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=1dba1206345c)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656154, encodeId=5eab165615417, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=FASEB：EGCG通过调节IRS / AKT和ERK / CREB / BDNF来改善高脂肪和高糖诱导的认知缺陷, objectType=article, longId=108803, objectId=1f8e108803a5, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=1f8e108803a5, replyNumber=0, likeNumber=72, createdTime=2018-02-28, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=1f8e108803a5, moduleTitle=FASEB：EGCG通过调节IRS / AKT和ERK / CREB / BDNF来改善高脂肪和高糖诱导的认知缺陷, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=1f8e108803a5)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656156, encodeId=f07d1656156c0, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Free Radic Biol Med ：UCP2与Akt/mTOR通路联合作用有作为胰腺腺癌治疗的新型治疗策略潜力, objectType=article, longId=117967, objectId=bdfc11e9676b, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=bdfc11e9676b, replyNumber=0, likeNumber=76, createdTime=2017-11-14, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=bdfc11e9676b, moduleTitle=Free Radic Biol Med ：UCP2与Akt/mTOR通路联合作用有作为胰腺腺癌治疗的新型治疗策略潜力, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=bdfc11e9676b)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656166, encodeId=0b0016561660e, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Diabetologia：在1型糖尿病小鼠模型中膜联蛋白A1通过恢复Akt信号传导来减弱微血管并发症, objectType=article, longId=136469, objectId=30c41364691d, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=30c41364691d, replyNumber=0, likeNumber=61, createdTime=2018-09-21, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=30c41364691d, moduleTitle=Diabetologia：在1型糖尿病小鼠模型中膜联蛋白A1通过恢复Akt信号传导来减弱微血管并发症, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=30c41364691d)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656157, encodeId=887b165615e11, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Diabetes：新发现一个AKT2的功能丧失性突变可降低多种组织的胰岛素刺激性糖摄取。, objectType=article, longId=120444, objectId=b8a212044441, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=b8a212044441, replyNumber=0, likeNumber=75, createdTime=2018-09-17, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=b8a212044441, moduleTitle=Diabetes：新发现一个AKT2的功能丧失性突变可降低多种组织的胰岛素刺激性糖摄取。, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=b8a212044441)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656164, encodeId=8270165616469, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=JCEM：Akt2突变可导致血脂代谢异常者出现低血糖, objectType=article, longId=131300, objectId=13d313130066, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=13d313130066, replyNumber=0, likeNumber=0, createdTime=2018-05-14, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=13d313130066, moduleTitle=JCEM：Akt2突变可导致血脂代谢异常者出现低血糖, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=13d313130066)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656165, encodeId=3e47165616538, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=PLoS One：蛇床子素可通过调节PI3K/AKT抑制胃癌细胞的增殖, objectType=article, longId=135290, objectId=3d5c13529053, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=3d5c13529053, replyNumber=0, likeNumber=73, createdTime=2018-10-20, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=3d5c13529053, moduleTitle=PLoS One：蛇床子素可通过调节PI3K/AKT抑制胃癌细胞的增殖, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=3d5c13529053)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656152, encodeId=6b7c165615213, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=SCI REP：RASD1的过表达抑制胶质瘤细胞迁移/侵袭，并使AKT / mTOR信号通路失活, objectType=article, longId=101507, objectId=ba7e10150e84, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=ba7e10150e84, replyNumber=0, likeNumber=78, createdTime=2018-03-02, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=ba7e10150e84, moduleTitle=SCI REP：RASD1的过表达抑制胶质瘤细胞迁移/侵袭，并使AKT / mTOR信号通路失活, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=ba7e10150e84)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656161, encodeId=4c82165616191, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=Stem cells：激活AKT-mTOR信号，可促进间充质干细胞的肌腱发生过程。, objectType=article, longId=126592, objectId=3079126592c5, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=3079126592c5, replyNumber=0, likeNumber=96, createdTime=2018-07-31, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=3079126592c5, moduleTitle=Stem cells：激活AKT-mTOR信号，可促进间充质干细胞的肌腱发生过程。, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=3079126592c5)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656150, encodeId=189f165615066, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=J CLIN INVEST：AKT介导的组蛋白甲基转移酶WHSC1稳态促进前列腺癌转移, objectType=article, longId=93060, objectId=13e89306022, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=13e89306022, replyNumber=0, likeNumber=59, createdTime=2017-11-09, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=13e89306022, moduleTitle=J CLIN INVEST：AKT介导的组蛋白甲基转移酶WHSC1稳态促进前列腺癌转移, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=13e89306022)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1656149, encodeId=7297165614919, content=<a href='/topic/show?id=f4782246ec' target=_blank style='color:#2F92EE;'>#Akt#</a>, objectTitle=PNAS：AKT对记忆T细胞的生存非常关键，有望用于肿瘤免疫治疗, objectType=article, longId=88282, objectId=97ca8828218, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=97ca8828218, replyNumber=0, likeNumber=0, createdTime=2017-08-01, rootId=0, userName=xfpan20, userId=b8c724776724, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=97ca8828218, moduleTitle=PNAS：AKT对记忆T细胞的生存非常关键，有望用于肿瘤免疫治疗, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=97ca8828218)], followStatus=false, userIsMember=false, type=1, lengthMark=0)] 梅斯话题小助手

Akt

2020-05-29

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

J Periodontol：TLR激活通过Akt信号通过Myd88或TRIF依赖性方式抑制人牙周膜干细胞的成骨潜能

2019-07-30

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Clin Exp Pharmacol Physiol：MicroRNA let-7c-5p通过HMGA2/PI3K/Akt信号阻断抑制脂多糖诱导的炎症

2019-01-09

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

J Cell Physiol：细胞外酶促合成的糖原通过Akt / GSK-3β信号传导途径激活成骨细胞分化来促进成骨

2019-09-02

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Cell Physiol Biochem：IL-8通过CXCR2介导的PI3k/Akt信号通路增强BMSCs的骨再生作用

2018-12-28

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

J Clin Invest：PTEN/AKT和Hippo/YAP/TAZ通路之间的交叉串扰加速了NAFLD和肝癌的发生

2018-04-01

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Blood：IGF-1通过激活Akt促进血小板生成

2018-07-27

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Prostate Cancer and P D：生殖细胞中 IL4、MGMT和AKT1变异与前列腺癌特异性死亡率相关

2018-11-10

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Int J Oncol：金-大黄酚纳米颗粒可通过失活AKT并诱导细胞凋亡和ROS产生抑制前列腺癌的进展

2018-07-26

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

CLIN CANCER RES：TMCO1影响膀胱尿路上皮癌AKT信号通路

2018-12-03

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

CLIN CANCER RES：AKT抑制剂MK-2206联合阿那曲唑治疗Ⅱ/Ⅲ期PIK3CA突变ER阳性和HER-2阴性乳腺癌

2017-12-15

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

FASEB：EGCG通过调节IRS / AKT和ERK / CREB / BDNF来改善高脂肪和高糖诱导的认知缺陷

2018-02-28

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Free Radic Biol Med ：UCP2与Akt/mTOR通路联合作用有作为胰腺腺癌治疗的新型治疗策略潜力

2017-11-14

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Diabetologia：在1型糖尿病小鼠模型中膜联蛋白A1通过恢复Akt信号传导来减弱微血管并发症

2018-09-21

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Diabetes：新发现一个AKT2的功能丧失性突变可降低多种组织的胰岛素刺激性糖摄取。

2018-09-17

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

JCEM：Akt2突变可导致血脂代谢异常者出现低血糖

2018-05-14

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

PLoS One：蛇床子素可通过调节PI3K/AKT抑制胃癌细胞的增殖

2018-10-20

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

SCI REP：RASD1的过表达抑制胶质瘤细胞迁移/侵袭，并使AKT / mTOR信号通路失活

2018-03-02

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

Stem cells：激活AKT-mTOR信号，可促进间充质干细胞的肌腱发生过程。

2018-07-31

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

J CLIN INVEST：AKT介导的组蛋白甲基转移酶WHSC1稳态促进前列腺癌转移

2017-11-09

xfpan20 (审核中...)

前往app查看评论内容

#Akt#

PNAS：AKT对记忆T细胞的生存非常关键，有望用于肿瘤免疫治疗

2017-08-01

科室
- - 订阅+
  - 更多科室
工具
服务